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It is important to understand that krill oil has a number of other advantages over fish oil:
Some studies have shown that krill oil may be 48 times more potent than fish oil. This means you need far less of it than fish oil, as confirmed by a 2011 study published in the journal Lipidsii.
This is because krill oil contains phospholipids, so the omega-3 fats are already in the form that your body can use. This bioavailability means krill oil is absorbed very quickly and crosses your blood-brain barrier, so is able to reach important brain structures. Also, phospholipids are one of the principle compounds in high-density lipoproteins (HDL), which you want more of.
Fish oil is quite prone to oxidation, and oxidation leads to the formation of free radicals. Consuming free radicals further increases your need for antioxidants. Fish oil is weak in antioxidant content, whereas krill oil is rich in antioxidants. Krill oil contains astaxanthin—probably the most potent antioxidant in nature—which is why it is so stable and resistant to oxidation.
Many, if not most, fish and fish oil are now contaminated with mercury and other heavy metals; even fish that is thousands of miles away from coal plants and other environment-polluting industries. Antarctic krill is not prone to this contamination.
Krill is also far more sustainable as a food source than is fish because it's the largest biomass in the world, making krill harvesting one of the most sustainable practices on the planet.
The only kind of krill oil I recommend is from genuine Antarctic krill. Look for a brand that is cold-processed, which preserves its biological benefits. Please make sure that hexane is not used to extract the oil from the krill as some of the most popular krill oils on the market use this dangerous chemical agent. It should also be free of heavy metals, PCBs, dioxins and other contaminants. You should also make sure the krill you take is harvested in compliance with international conservation standards.
Vitamin D has sometimes been regarded as the most potentially dangerous vitamin. In his 2001 article "Vitamin Toxicity," Mark Rosenbloom, MD, writes that, for vitamin D, "Acute toxic dose is not established, and chronic toxic dose is more than 50,000 IU/day in adults. In children, 400 IU/day is potentially toxic. A wide variance in potential toxicity exists." There were no fatalities cited.
Vitamin D has an important role in cardiovascular health. For example, not only can it prevent hypertension, it can help treat it. "Hypertension appears to improve with vitamin D supplementation whether or not the vitamin is deficient." This is an important point.
Congestive heart failure (CHF) may be caused by vitamin D deficiency. "Low vitamin D status can explain alterations in mineral metabolism as well as myocardial dysfunction in the CHF patients, and it may therefore be a contributing factor in the pathogenesis of CHF." Not surprisingly, bone loss is associated with congestive heart failure. Dilated cardiomyopathy has been linked with rickets, both of which "responded well to supplemental calcium and vitamin D." )
diopathic dilated cardiomyopathy is a clinical diagnosis. After exclusion of all identifiable causes (except genetic), DCM is traditionally referred to as idiopathic dilated cardiomyopathy. It is important to note that because the term idiopathic dilated cardiomyopathy was used before evidence of genetic forms emerged, the diagnosis of idiopathic dilated cardiomyopathy does not differentiate between genetic and non-genetic causes; therefore, a portion of individuals with ‘idiopathic dilated cardiomyopathy’ may have genetic forms.
The most common cause of DCM (in which the term DCM is used generically to describe the morphology and function of the left ventricle regardless of etiology) is ischemic injury, such as that caused by coronary artery disease or prior myocardial infarction.
After ischemic injury, other common causes of DCM include valvular and congenital heart disease, toxins (e.g., anthracyclines), thyroid disease, inflammatory conditions, myocarditis, severe long-standing hypertension, and radiation. Most of these can be detected with a careful medical history, a targeted physical examination, results of laboratory testing, an echocardiogram and, if indicated, coronary angiography to exclude coronary heart disease.
When each of two or more closely related family members meet a formal diagnostic standard for idiopathic dilated cardiomyopathy (i.e., all detectable causes of DCM, except genetic, have been ruled out), the diagnosis of familial dilated cardiomyopathy (FDC) is made.
Current evidence indicates that idiopathic dilated cardiomyopathy may be familial (and therefore possibly genetic) in approximately 20%-35% of cases.